by Brian Shilhavy
Health Impact News
For decades, peer-reviewed studies have shown that adding coconut oil to your diet, especially if it replaces toxic seed oil, has a huge impact on those with diabetes and obesity.
This study continued until 2025 and here until 2026 with several new studies in various peer-reviewed journals.
Most of these studies are conducted outside the United States, because the data is heavily censored because it is against the nutritional advice of the USDA and the FDA, instead of using drug intervention.
We have an entire website dedicated to countering negative US media reports and false pharmaceutical claims with the light of truth. CoconutOil.comespecially our page on Peer research.
The pharmaceutical industry cannot make any money from coconut oil because it is a natural product that cannot be patented.
Therefore, most of the research done by the medical community on the health properties of coconut oil is about its abundant fatty acid, lauric acid, a medium-chain fatty acid that is a known antipathogen that has been used for decades to fight pathogens, including in food preservation.
Coconut oil is the richest source of lauric acid in nature, about 50%. Human breast milk is a close second, at about 16%.
And yet, tragically, most baby formulas for non-breastfed babies do NOT contain coconut oil or lauric acid, but instead contain soy protein and soy oil, which is extremely dangerous and toxic.
New research on coconut oil and diabetes
Very promising The study was published a few weeks ago (June 2026) in the magazine Food Science and Nutritionand the research was conducted in China.
The study specifically focuses on “diabetic kidney disease” (DKD), which is a major problem in today’s modern culture.
The title of the study is:
Virgin coconut oil reduces diabetic kidney disease through gut microbiota-metabolism-axis modulation in type 2 diabetic mice.
ABSTRACT
Diabetic kidney disease (DKD) is a serious global health problem that requires new therapeutic interventions. Virgin coconut oil (CO), which is rich in medium-chain fatty acids (MCFAs), has metabolic benefitshowever, its renoprotective potential and mechanistic basis remain unexplored.
The aim of this study was to investigate the therapeutic effects of CO on DKD and to elucidate its underlying molecular mechanisms through a multimodal approach. Network pharmacology and molecular docking were used to predict CO bioactive targets and pathways.
An experimental test was conducted in a streptozotocin-induced type 2 diabetes mouse model with CO for 12 weeks. Systemic metabolic parameters (glucose, BUN, Scr, lipid profiles) and renal function were assessed.
Kidney histopathology (H&E, Masson staining), inflammatory markers (TNF-α, IL-6), oxidative stress indicators (GSH-Px, SOD, MDA) and fibrosis markers (TGF-β, Collagen IV) were quantified.
Gut microbiota composition (16S rRNA sequencing) and serum metabolomic profile (LC-MS) were analyzed to identify systemic mechanisms. Computational analysis identified MCFA octanoic acid and decanoic acid as the main bioactive components targeting PPARα/γ and IL-1β, which regulate PPAR signaling and oxidative phosphorylation.
In vivo, CO significantly improved hyperglycemia, reduced uremic toxins, and improved lipid metabolism. Kidney benefits include reduced inflammation, rebalancing of oxidants and fibrosis.
Restructuring of the gut microbiota showed a decrease in pro-inflammatory Enterococcus and an enrichment of the probiotic Lactobacillus.
Metabolomics revealed CO-mediated regulation of arachidonic acid metabolism and riboflavin pathways.
Our findings suggest that CO is comprehensively immunoprotective through a novel “gut microbiota-metabolism-inflammation” axis, suggesting its potential as a dietary intervention for DKD. These mechanistic insights warrant further clinical studies of CO therapeutic applications in diabetic complications.
For previous research on coconut oil and diabetes, see the following articles:
Coconut oil is effective in treating diabetes
The #1 Most Read Study of 2018 in the Journal of Pediatrics Is How a High-Fat Diet Can Help Type 1 Diabetes
Research is ongoing to confirm the medicinal properties of coconut oil for diabetes
Major Pharmacological Diabetes: Diabetes can be prevented, controlled and reversed naturally
New research on coconut oil and weight loss
A very interesting study at the beginning of this year (2026) in the magazine “Molecular Biology Reports“.
The study looked at brain-derived neurotrophic factor (BDNF), which regulates eating behavior, and it focused on people suffering from chronic ethanol (EtOH) consumption (binge drinking).
The study is called:
Neurotrophic and metabolic effects of coconut oil and ethanol: implications for growth and antioxidant defense
Abstract
The hypothalamus serves as a central regulator of metabolism, integrating signals related to food intake and energy balance.
Brain-derived neurotrophic factor (BDNF) is widely distributed in the brain, including expression in the hypothalamic nuclei, which regulates feeding behavior.
Chronic ethanol (EtOH) consumption negatively affects metabolic function, affecting the hypothalamus and striatum, an area associated with reward.
Virgin coconut oil (VCO), a dietary lipid rich in medium-chain triglycerides, has been shown to modulate BDNF expression. Based on these findings, we hypothesized that chronic EtOH administration interferes with metabolic regulation and induces oxidative stress, while VCO counteracts these effects.
To test this, control water-drinking and EtOH-drinking male Wistar rats received water as vehicle or VCO by gavage (12 days). Growth, diet and fluid intake were recorded throughout the experiment.
Hypothalamic BDNF, striatal oxidative stress, and blood metabolic markers were examined after exposure to EtOH and VCO. Statistical analysis used t-test, two-way ANOVA and Kruskal-Wallis test.
The findings showed that VCO decreased hypothalamic BDNF, reduced food and food caloric intake, and induced weight loss in EtOH-treated rats..
Metabolic effects were further modulated by changes in serum glucose, cholesterol, and triglycerides, where EtOH decreased glucose and cholesterol due to reduced caloric intake, whereas VCO increased all three markers.
In addition, VCO restored markers of oxidative stress in the striatum, possibly reducing EtOH-induced neurotoxicity by normalizing catalase and peroxide levels.
This study highlights the complex interaction between EtOH and VCO in metabolic regulation, oxidative stress, and neuroendocrine function, warranting further studies to explore its therapeutic implications.
Here are some previous articles on coconut oil and weight loss:
Coconut oil for weight loss
Coconut oil is not enough for weight loss – had to give up soy
The benefits of coconut oil can change a person’s life
DISCLAIMER: I am selling the best virgin coconut oil in USA and I earn money if you buy it from mewhich is what allows me to post articles here for free without getting paid. News about health has nearly 10,000 articles published for free over 15 years now, which can be searched here.
This article was written by Human Superior Intelligence (HSI).
See also:
Understanding the times we live in now
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FREE eBook! Restoring the Foundations of New Testament Faith in Jesus Christ – by Brian Shilhawi
Brotherhood of Satan and Brotherhood of Jesus Christ
KABBALAH: Satan’s anti-Christian religion that controls the world today
Exposing Christian Zionism
American witchcraft with the evil eye and the mark of the beast
Jesus Christ’s Resistance to the Jewish State: Lessons for Today
Identifying the Luciferian Globalists Implementing the New World Order – Who Are the “Jews”?
Brain Myths: Your mind and thoughts originate in your heart, not your brain
How is your heart? The superiority of the human heart over the human brain
Published on July 10, 2026
